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Home Physician Enquiries

Physcian Enquiries

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Wave Brilliance Australia is  a distributor and supplier of the ExMI pelvic floor rehabilitation system in Australia and New Zealand, a corporate member of the Continence Foundation of Australia, R.A.C.G.P. Accredited Provider and member of the Prostate Cancer Foundation of Australia.

Our Mission Statement - A vision to see, the power to treat

Empowering GP's to treat their patients for disorders of the pelvic floor

At last, a highly effective, affordable treatment option is available at primary care level. We encourage all GP's to evaluate the benefits of ExMI and include this approach into their practice. Please click here to arrange a copy of purchase & rental options


The Role of Extracorporeal Magnetic Innervation (ExMI™)

In the Treatment of Urinary Incontinence

 

Class Type Patho-physiology Functional Defect or Level of Severity Does ExMI have a role?

Decreased Urethral Resistance

Anatomic Support Defects (GSI I/II)

Failure of adequate urethral closure

Support Defect

Yes

(Stress)

Greater forces on posterior vs. anterior wall of urethra

Anatomic Defect

No

Functional Support

Yes

Intrinsic Sphincter Deficiency (ISD)

Deficiency in urethral closure mechanism

Mild

Yes

Moderate

Yes

Severe

Probably Not

Failure to Inhibit the Detrusor

Failure to contract pelvic floor releases detrusor reflex

Neurogenic – Mild / Moderate

Yes

Neurogenic – Severe

No

Behavioral

Yes

Mechanical

Probably Not

Increased Urethral Resistance (Overflow / Retention)

Anatomic Obstruction

Urethral compression or narrowing

No

Functional Obstruction

Failure of normal relaxation

Neurogenic

Yes

Behavioral (failure to relax external sphincter)

Yes

Inhibition of Detrusor Activity

Failure to relax pelvic floor muscles inhibits detrusor ability to start and maintain a contraction

Neurogenic

Yes

Behavioral

Yes

Situational

Yes

Increased Intravesical Pressure (Urge)

Uninhibited Detrusor Contractions

Increased intravesical pressure overcomes urethral resistance

Overactive Bladder

Yes

Detrusor Hyper Reflexia

Yes

Decreased accommodation

Fibrosis

No

Neurogenic

No

Decreased Intravesical Pressure (Overflow / Retention)

Peripheral Denervation or Neuropathy

Absent neuro input

Congenital

No

Diabetics

Perhaps

Detrusor Myopathy

Smooth muscle damage

Fibrosis

No

Inflammation

No